mouse tgf-beta 1 duoset elisa Search Results


tgf β dy1679  (R&D Systems)
96
R&D Systems tgf β dy1679
TGF- β (A), IL-1 β (B), IL-6 (C), TNF- α (D), IL-13 (E), and IL-4 (F) levels in lung homogenates Bar and line graphs present mean ± standard error. Cht, chitin; CPX, chitinase-1 inhibitor; OVA, ovalbumin * P < 0.05 between them analyzed by one-way ANOVA followed by post-hoc Bonferroni test.
Tgf β Dy1679, supplied by R&D Systems, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/mouse+tgf-beta+1+duoset+elisa/Mouse+TGF-beta+1+DuoSet+ELISA/pmc12034102-65-32-47
Average 96 stars, based on 1 article reviews
tgf β dy1679 - by Bioz Stars, 2026-10
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mouse tgf β 1 duoset elisa  (R&D Systems)
96
R&D Systems mouse tgf β 1 duoset elisa
A) UMAP analysis of reclustered alveolar, transitional/inflammatory, and fibrotic fibroblasts with superimposed vector from pseudotime trajectory analysis reveals no Ptgfr dependent effect on terminal node. (B) UMAP analysis of reclustered adventitial, transitional/inflammatory, and fibrotic fibroblasts with superimposed vector from pseudotime trajectory analysis demonstrates a Ptgfr dependent effect on terminal node. In I ER - Sftpc I 73 T / Ptgfr − / − the terminal node is found in the transitional/inflammatory cluster while I ER - Sftpc I 73 T / Ptgfr +/+ samples have a vector terminating in the fibrotic cluster. (C) Comparative analysis of gene expression within the fibrotic cluster of I ER - Sftpc I 73 T / Ptgfr +/+ and I ER - Sftpc I 73 T / Ptgfr − / − mice is presented by gradient gene expression dot plots. Marker genes associated with the transitional/inflammatory cluster are comparatively elevated in I ER - Sftpc I 73 T / Ptgfr − / − mice while fibrotic marker genes are elevated in the I ER - Sftpc I 73 T / Ptgfr +/+ mice. (D) KEGG pathway enrichment analysis comparing the fibrotic clusters identifies multiple pathways associated with cytoskeletal rearrangement, mesenchymal activation, and <t>TGF</t> β signaling that are upregulated in I ER - Sftpc I 73 T / Ptgfr +/+ mice. (E) Measurement of BALF TGF β 1 via <t>elisa</t> demonstrates a significant decrease in I ER - Sftpc I 73 T / Ptgfr − / − mice (n=18) as compared to I ER - Sftpc I 73 T / Ptgfr +/+ mice (n=18). *p < 0.05
Mouse Tgf β 1 Duoset Elisa, supplied by R&D Systems, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/mouse+tgf-beta+1+duoset+elisa/Mouse+TGF-beta+1+DuoSet+ELISA/bio_rxiv__2023__06__07__543956-57-11-17
Average 96 stars, based on 1 article reviews
mouse tgf β 1 duoset elisa - by Bioz Stars, 2026-10
96/100 stars
  Buy from Supplier

Image Search Results


TGF- β (A), IL-1 β (B), IL-6 (C), TNF- α (D), IL-13 (E), and IL-4 (F) levels in lung homogenates Bar and line graphs present mean ± standard error. Cht, chitin; CPX, chitinase-1 inhibitor; OVA, ovalbumin * P < 0.05 between them analyzed by one-way ANOVA followed by post-hoc Bonferroni test.

Journal: Life sciences

Article Title: Effects of chitinase-1 inhibitor in obesity-induced and -aggravated asthma in a murine model

doi: 10.1016/j.lfs.2023.122163

Figure Lengend Snippet: TGF- β (A), IL-1 β (B), IL-6 (C), TNF- α (D), IL-13 (E), and IL-4 (F) levels in lung homogenates Bar and line graphs present mean ± standard error. Cht, chitin; CPX, chitinase-1 inhibitor; OVA, ovalbumin * P < 0.05 between them analyzed by one-way ANOVA followed by post-hoc Bonferroni test.

Article Snippet: The concentrations of chitinase-1 (Fine biotech, EM0939, Wuhan, China), hydroxyproline (Biovision, K555–100, CA, USA), collagen-1 (Fine biotech, EM6446), interleukin (IL)-1 β (DY401), TNF- α (DY410), IL-4 (DY404), IL-6 (DY406), IL-13 (DY413), and TGF- β (DY1679) in lung homogenates were assessed using an enzyme-linked immunosorbent assay (ELISA) kit (R&D Systems, San Diego, USA) according to the manufacturer’s instructions.

Techniques:

Pathologic findings (H&E, PAS, and MT; all × 200 magnification) (A), quantitative fibrosis area (B), level of hydroxyproline in lung homogenates (C), and level of collagen-1 in lung homogenates (D). TGF- β (E), TNF- α (F), IL-1 β (G), IL-6 (H), IL-4 (I), and IL-13 (J) levels in lung homogenates Bar and line graphs present mean ± standard error. Cht, chitin; CPX, chitinase-1 inhibitor; HFD, high-fat diet; OVA, ovalbumin; H&E, hematoxylin and eosin; PAS, periodic acid-Schiff; MT, Masson’s trichrome * P < 0.05 between them analyzed by one-way ANOVA followed by post-hoc Bonferroni test.

Journal: Life sciences

Article Title: Effects of chitinase-1 inhibitor in obesity-induced and -aggravated asthma in a murine model

doi: 10.1016/j.lfs.2023.122163

Figure Lengend Snippet: Pathologic findings (H&E, PAS, and MT; all × 200 magnification) (A), quantitative fibrosis area (B), level of hydroxyproline in lung homogenates (C), and level of collagen-1 in lung homogenates (D). TGF- β (E), TNF- α (F), IL-1 β (G), IL-6 (H), IL-4 (I), and IL-13 (J) levels in lung homogenates Bar and line graphs present mean ± standard error. Cht, chitin; CPX, chitinase-1 inhibitor; HFD, high-fat diet; OVA, ovalbumin; H&E, hematoxylin and eosin; PAS, periodic acid-Schiff; MT, Masson’s trichrome * P < 0.05 between them analyzed by one-way ANOVA followed by post-hoc Bonferroni test.

Article Snippet: The concentrations of chitinase-1 (Fine biotech, EM0939, Wuhan, China), hydroxyproline (Biovision, K555–100, CA, USA), collagen-1 (Fine biotech, EM6446), interleukin (IL)-1 β (DY401), TNF- α (DY410), IL-4 (DY404), IL-6 (DY406), IL-13 (DY413), and TGF- β (DY1679) in lung homogenates were assessed using an enzyme-linked immunosorbent assay (ELISA) kit (R&D Systems, San Diego, USA) according to the manufacturer’s instructions.

Techniques:

A) UMAP analysis of reclustered alveolar, transitional/inflammatory, and fibrotic fibroblasts with superimposed vector from pseudotime trajectory analysis reveals no Ptgfr dependent effect on terminal node. (B) UMAP analysis of reclustered adventitial, transitional/inflammatory, and fibrotic fibroblasts with superimposed vector from pseudotime trajectory analysis demonstrates a Ptgfr dependent effect on terminal node. In I ER - Sftpc I 73 T / Ptgfr − / − the terminal node is found in the transitional/inflammatory cluster while I ER - Sftpc I 73 T / Ptgfr +/+ samples have a vector terminating in the fibrotic cluster. (C) Comparative analysis of gene expression within the fibrotic cluster of I ER - Sftpc I 73 T / Ptgfr +/+ and I ER - Sftpc I 73 T / Ptgfr − / − mice is presented by gradient gene expression dot plots. Marker genes associated with the transitional/inflammatory cluster are comparatively elevated in I ER - Sftpc I 73 T / Ptgfr − / − mice while fibrotic marker genes are elevated in the I ER - Sftpc I 73 T / Ptgfr +/+ mice. (D) KEGG pathway enrichment analysis comparing the fibrotic clusters identifies multiple pathways associated with cytoskeletal rearrangement, mesenchymal activation, and TGF β signaling that are upregulated in I ER - Sftpc I 73 T / Ptgfr +/+ mice. (E) Measurement of BALF TGF β 1 via elisa demonstrates a significant decrease in I ER - Sftpc I 73 T / Ptgfr − / − mice (n=18) as compared to I ER - Sftpc I 73 T / Ptgfr +/+ mice (n=18). *p < 0.05

Journal: bioRxiv

Article Title: Disruption of Prostaglandin F 2α Receptor Signaling Attenuates Fibrotic Remodeling and Alters Fibroblast Population Dynamics in A Preclinical Murine Model of Idiopathic Pulmonary Fibrosis

doi: 10.1101/2023.06.07.543956

Figure Lengend Snippet: A) UMAP analysis of reclustered alveolar, transitional/inflammatory, and fibrotic fibroblasts with superimposed vector from pseudotime trajectory analysis reveals no Ptgfr dependent effect on terminal node. (B) UMAP analysis of reclustered adventitial, transitional/inflammatory, and fibrotic fibroblasts with superimposed vector from pseudotime trajectory analysis demonstrates a Ptgfr dependent effect on terminal node. In I ER - Sftpc I 73 T / Ptgfr − / − the terminal node is found in the transitional/inflammatory cluster while I ER - Sftpc I 73 T / Ptgfr +/+ samples have a vector terminating in the fibrotic cluster. (C) Comparative analysis of gene expression within the fibrotic cluster of I ER - Sftpc I 73 T / Ptgfr +/+ and I ER - Sftpc I 73 T / Ptgfr − / − mice is presented by gradient gene expression dot plots. Marker genes associated with the transitional/inflammatory cluster are comparatively elevated in I ER - Sftpc I 73 T / Ptgfr − / − mice while fibrotic marker genes are elevated in the I ER - Sftpc I 73 T / Ptgfr +/+ mice. (D) KEGG pathway enrichment analysis comparing the fibrotic clusters identifies multiple pathways associated with cytoskeletal rearrangement, mesenchymal activation, and TGF β signaling that are upregulated in I ER - Sftpc I 73 T / Ptgfr +/+ mice. (E) Measurement of BALF TGF β 1 via elisa demonstrates a significant decrease in I ER - Sftpc I 73 T / Ptgfr − / − mice (n=18) as compared to I ER - Sftpc I 73 T / Ptgfr +/+ mice (n=18). *p < 0.05

Article Snippet: TGF β concentration in the cell free BALF was calculated using Mouse TGF β 1 DuoSet Elisa (R&D Systems Cat DY1679-05) according to the manufacturer’s instructions.

Techniques: Plasmid Preparation, Gene Expression, Marker, Activation Assay, Enzyme-linked Immunosorbent Assay